Acute Oxygen Toxin, Chronic Oxygen Toxin, Factors Influencing Oxygen Toxin, Effects of Oxygen Toxin on Histopathology, Complications of Oxygen Toxin, Evaluation of Oxygen Toxin, Diagnosis of Oxygen Toxin And Treatment of Oxygen Toxin
Creators
- 1. Veterinay Doctor, Shantiram medical college Nandyal, Andhra Pradesh, India
- 2. Staff IVRI UP, College of Veterinary Science, Rajendra Nagr, Telengana, India
- 3. Assistant General Manager, Vaishnavi Bio pharma pvt. Ltd,Telanagana, India
- 4. Contract Teaching Faculty, College of veterinary science, Proddatur, Andhra Pradesh, India
- 5. Assistant Professor, College of Veterinary Science, Gopavaram, Proddatur, Andhra Pradesh, India
- 6. Associate Professor, College of Veterinary Science, Proddatur, Andhra Pradesh, India
Description
The most often used medication in hospitals is oxygen, which is a crucial component in prehospital treatment. Prehospital providers administer oxygen to eliminate hypoxemia and hypoxia. Clinical settings of oxygen toxicity are categorized into acute oxygen toxin and habitual (chronic) oxygen toxin. Oxygen toxin is based on time of exposure, hyperbaric hypoxia, normobaric hypoxia, Fi)2 more than 60 longer than 36 hours. Oxygen toxin effects on histopathology are related to pulmonary edema, pulmonary injury and intra-alveolar hemorrhage. Complications of oxygen toxin are disorientation, breathing problems and visual changes similar to diplopia as well as cataract confirmation. Evaluation of oxygen toxin is estimated by pulmonary function tests and chest X-ray which can exhibit signs of respiratory distress syndrome (ARDS). Reducing exposure to high-oxygen situations is a key component of managing oxygen poisoning. Regarding treatment, exogenous antioxidants, particularly vitamin C and E have been more useful to decrease the frequency of retro lental fibroplasia in unseasonal babies on hyperoxic remedy.
Files
ACUTE OXYGEN TOXIN -Formatted Paper Paper.pdf
Files
(302.3 kB)
| Name | Size | Download all |
|---|---|---|
|
md5:099220ce8921f43a0bee023b35ca32ef
|
302.3 kB | Preview Download |
Additional details
References
- 1. Hafner, S., Beloncle, F., Koch, A., Radermacher, P., & Asfar, P. (2015). Hyperoxia in intensive care, emergency, and peri-operative medicine: Dr. Jekyll or Mr. Hyde? A 2015 update. Annals of intensive care, 5, 1-14.
- 2. Thomson, L., & Paton, J. (2014). Oxygen toxicity. Paediatric respiratory reviews, 15(2), 120-123.
- 3. Camporesi, E. M. (2014). Side effects of hyperbaric oxygen therapy. Undersea & hyperbaric medicine: journal of the Undersea and Hyperbaric Medical Society, Inc, 41(3), 253-257.
- 4. Banharn, N. D. (2011). Oxygen toxicity seizures: 20 years' experience from a single hyperbaric unit.
- 5. Matalon, S., Nesarajah, M. S., & Farhi, L. E. (1982). Pulmonary and circulatory changes in conscious sheep exposed to 100% O2 at 1 ATA. Journal of Applied Physiology, 53(1), 110-116.
- 6. Brock, T. G., & Giulio, C. D. (2006). Prolonged exposure to hyperoxia increases perivascular mast cells in rat lungs. Journal of Histochemistry & Cytochemistry, 54(11), 1239-1246.
- 7. Wingelaar, T. T., Van Ooij, P. J. A., Brinkman, P., & Van Hulst, R. A. (2019). Pulmonary oxygen toxicity in navy divers: a crossover study using exhaled breath analysis after a one-hour air or oxygen dive at nine meters of sea water. Frontiers in Physiology, 10, 10.
- 8. Chawla, A., & Lavania, A. K. (2001). Oxygen toxicity. Medical Journal Armed Forces India, 57(2), 131-133.
- 9. Domachevsky, L., Rachmany, L., Barak, Y., Rubovitch, V., Abramovich, A., & Pick, C. G. (2013). Hyperbaric oxygen-induced seizures cause a transient decrement in cognitive function. Neuroscience, 247, 328-334.
- 10. Farmery, S., & Sykes, O. (2012). Neurological oxygen toxicity. Emergency Medicine Journal, 29(10), 851-852.