Published April 20, 2023
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Journal article
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Post-prandial increases in liver-gut hormone LEAP2 correlate with attenuated eating behaviour in adults without obesity (supplementary material)
- 1. PsychoNeuroEndocrinology Research Group, Division of Psychiatry, Department of Brain Sciences, Faculty of Medicine, Imperial College London, Hammersmith Hospital, London, W12 0NN, UK
- 2. School of Public Health, Faculty of Medicine, Imperial College London, St. Mary's Hospital, London, W2 1PG, UK
Description
Figure S1. Study Protocol
Figure S2. Correlation of post-prandial plasma LEAP2 with meal size consumed across different studies
Figure S3. Plasma LEAP2 at study visits
Figure S4. Food appeal ratings between visits for high-energy and low-energy foods
Table S1. Whole brain analysis for correlations of post-prandial changes in BOLD signal to high-energy foods with plasma hormones
Table S2. Whole brain analysis for correlations of post-prandial changes in BOLD signal to low-energy foods with plasma hormones
Table S3. Whole brain analysis for correlations of post-prandial changes in BOLD signal to high- or low-energy foods with plasma hormones
Supplementary References
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Bhargava_2023_J_Endocrine_Society_supplementary_material.pdf
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Additional details
Funding
- Human experimental medicine studies of gut hormone LEAP2, an endogenous ghrelin antagonist and target for obesity, diabetes, cachexia and addiction MR/T017279/1
- UK Research and Innovation