Published September 26, 2022 | Version v1
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Epigenome-Wide Meta-Analysis Reveals Associations Between Dietary Glycemic Index and Glycemic Load and DNA Methylation in Children and Adolescents of Different Body Sizes

Description

Diets high in glycemic index (GI) and glycemic load (GL) have been associated with increased risk for the development of several non-communicable diseases (NCDs) in children and in adults. DNA methylation changes may underlie these associations, in particular in at-risk groups, e.g., people with higher body-mass-index. We performed meta-analysis of epigenome-wide associations studies (EWAS) between dietary GI and GL and blood DNA methylation of 1,187 children and adolescents (age range: 4.5-17 years) from six studies. We also performed startified analysis for these exposures in people with normal-weight (n=801) or overweight/obesity (n=386). To adress potential inflation and bias in the individual EWAS analysis and meta-analysis, we used Bacon adjustment (bacon Bioconductor package) and the reported model estimates and standard errors are adjusted. Dietary GL was positively associated with DNAm of cg20274553 (FDR <0.05), annotated to WDR27. Several CpGs were identified in the normal-weight (GI: 85; GL: 17) and overweight or obese (GI: 136; GL: 298; FDR <0.05) strata. Follow-up analysis showed that in participants with overweight or obesity identified CpGs were related to RNA expression of genes associated with impaired metabolism (e.g., FRAT1, CSF3). Thus, high-GI and/or -GL diets may influence epigenetic gene regulation and thereby promote metabolic derangements in young people with increased BMI.

The corresponding paper can be found here: https://doi.org/10.2337/dc23-0474

 

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