Questioning the Zika Virus
Authors/Creators
Description
A growing body of health officials in Brazil are doubting that the Zika “virus” is
responsible for the rise in birth defects in parts of that country. Zika, along with
yellow fever, has been tossed into the family Flaviviruses; the Latin “flavus”
meaning yellow. But unlike yellow fever, the vast majority of Zika’s symptoms for
the last 70 years have been mild to non-existent. Despite disseminations by the lay
and scientific press, there are serious questions whether Zika causes microcephaly
at all. If by March, 2016 the Brazilian Ministry of Health reported 2,197 suspected
cases of microcephaly, only 11.48% of these were Zika-positive. Zika is widespread
throughout Brazil and South and Central America, yet the bulk of microcephaly
cases are confined to the costal tip of Northeastern Brazil. Furthermore, despite
extensive testing, no known mosquito-borne arbovirus or any other virus has to
this point been proven to cause Brazilian microcephaly.
While Zika was being portrayed as “the most alarming health crisis to hit Brazil
in decades”, tuberculosis and its related mycobacteria were quietly gaining a
stranglehold and building an ecologic niche in the very Northeastern region
being hit by epidemic microcephaly. Why was this important? With NE Brazilian
microcephaly/Zika we are probably dealing with a mosquito-fuelled environmental
zoonosis ― a disease that can be transmitted from animals to humans ― such
as primates, and to a lesser extent birds (Mycobacterium avium), and rodents
(Mycobacterium microti) , all mentioned in the Zika literature. Add to this the
penchant of Brazilian’s to illegally capture and keep mycobacterial-laden wild
monkeys and exotic birds as pets or for revenue, and you have a potential zoonotic
time-bomb ready to explode once the proper vectors presents themselves. Three
mosquito vectors have been steadily populating Northeastern Brazil: namely Culex
quinquefasciatus, the Aedes aegypti and the Aedes albopictus ― all of which have
the capacity to transmit viral-like forms of the mycobacteria associated with HIV
and through direct laboratory investigation with microcephaly. Perhaps it is time
to rethink what’s really behind Brazilian Microcephaly and other symptomatology
from the “Zika” agent.
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Questioning_the_Zika_Virus FS.pdf
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