反刍思维的机制重构假说 —— 基于 dACC‑DMS 环路冲突迭代模型

【中文摘要】
本文为两篇前期预印本的补遗工作,正式整合提出**认知‑行动卡顿闭环**病理心理框架。第一篇提出病理性反刍的效价冲突模型,聚焦dACC效价冲突引发的环路卡锁;第二篇提出时序迭代交付假说,解释DMS向DMS行为模板交付失败导致的行动卡顿。两篇原本独立,本文阐明二者耦合关系,补充焦虑介导的正反馈放大闭环。

本文完成一项范式翻转:传统观点认为病理性反刍的根源是负性内容的反复加工;本框架提出,病理性反刍本质是双向心理冲突持续激活dACC,驱动DMS进入无限迭代校验、无法定稿交付的神经环路卡顿现象,负性内容只是伴随产物,不是驱动根源。

文章区分两套子系统:A)ACC门控层卡顿,冲突无法消解时拒绝下发执行许可,表现为反复思虑、无法决断;B)DMS模拟‑编译层卡顿,缺少终结信号导致内部模板反复迭代而无法向下游固化交付,表现为反复推翻、无法收尾。焦虑作为耦合介质,经由HPA轴与杏仁核同时上调ACC冲突敏感度、收紧DMS迭代收敛阈值,形成自我强化三角正反馈循环。

基于该框架,干预重点不一定是改造思维内容或强制激活行为,而包含降低对卡顿本身的焦虑耐受、修复DMS‑DLS交付终止机制。文中给出4条可检验的实证预测,可供脑成像、生理与干预研究设计参考。本框架不替代现有理论,提供一套新的系统视角用于抑郁、反刍、强迫相关病理心理理解。

本系列使用的“交付、打磨、卡锁、审批”均为系统隐喻,对应皮层‑基底节‑丘脑环路突触权重重分配与门控重置,不是解剖实体隐喻。

【English Abstract】
This manuscript is a supplement to two prior preprints, formally integrating the **cognition‑action jam loop**, a unified psychopathological framework. The first preprint proposed the valence‑conflict model of pathological rumination, focusing on dACC‑triggered circuit locking under conflicting valence signals. The second preprint put forward the sequential iterative delivery hypothesis to explain action‑jam caused by failed template delivery from DMS to DLS. While originally published independently, the present work clarifies their coupling logic and adds the anxiety‑mediated positive‑feedback amplification loop.

A paradigm shift is proposed: classical theories attribute pathological rumination to repetitive processing of negative content. In this framework, pathological rumination arises when persistent bidirectional psychological conflicts activate dACC, forcing DMS into endless iterative validation with no conclusive delivery. Negative ideation and emotion are by‑products rather than primary drivers.

Two parallel subsystems are described: (A) ACC gating jam: unresolved conflict inhibits execution permission, manifested as rumination and indecision; (B) DMS simulation‑compilation jam: lacking termination signals, internal templates keep iterating without downstream consolidation, manifested as endless revision with no closure. Anxiety acts as a coupling mediator: via HPA‑axis and amygdala responses, it increases ACC sensitivity to conflict and tightens DMS convergence thresholds, forming a self‑reinforcing triangular positive‑feedback loop.

According to this model, interventions may target anxiety tolerance toward the jam state and restore DMS‑to‑DLS termination mechanisms, instead of merely restructuring thought content or forcing behavioral activation. Four testable empirical predictions are provided for neuroimaging, physiological and intervention studies. This framework does not replace existing theories, but offers a novel systematic perspective for understanding rumination‑related psychopathology in depression and obsessive‑compulsive spectrum conditions.

Terms such as “delivery”, “polishing”, “locking” and “approval” are system metaphors for synaptic weight reconfiguration and gating reset within cortico‑basalganglia‑thalamic circuits, not literal anatomical entities.

This is the third preprint of the author’s theoretical series. Two PDF files (Chinese and English versions) are available for download.

Related preprints:
① https://doi.org/10.5281/zenodo.21745928
② https://doi.org/10.5281/zenodo.21777450

Suggested citation:
[李维江 Weijiang Li]. (2026). 认知‑行动卡顿闭环:一个统一的病理心理框架(补遗). Preprint. Zenodo.