case_images: PMC9895116_01
case_images: Timeline of hospital course with significant findings each day.
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case_images: PMC9895116_01_fped-10-1058832-g001.jpg
case_images: F1
case_images: Since the creatinine and calcium initially improved with hydration and furosemide, it was assumed that the etiology of the AKI was a combination of prerenal (dehydration) and intrinsic (related to hypercalcemia) (see Figure 1).
case_images: Axial reformatted, lung window CT image through the lower chest demonstrating multiple noncalcified, well-demarcated pulmonary nodules throughout all lobes (black arrows).
case_images: fped-10-1058832-g002.jpg
case_images: PMC9895116_01_fped-10-1058832-g002.jpg
case_images: F2
case_images: Computed tomography (CT) revealed mediastinal lymphadenopathy that was granulomatous in nature and pulmonary nodules (see Figures 2, 3).
case_images: Coronal reformatted, soft-tissue window, CT image through the mediastinum with low attenuation, noncalcified, bihilar, and subcarinal adenopathy (white arrows).
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case_images: PMC9895116_01_fped-10-1058832-g003.jpg
case_images: F3
case_images: Computed tomography (CT) revealed mediastinal lymphadenopathy that was granulomatous in nature and pulmonary nodules (see Figures 2, 3).
case_images: AP portable chest radiograph with diffuse but lower lobe predominant micronodular opacities and air bronchograms.
case_images: fped-10-1058832-g004.jpg
case_images: PMC9895116_01_fped-10-1058832-g004.jpg
case_images: F4
case_images: Radiology noted that, in hindsight, there may have been findings consistent with histoplasmosis, specifically diffuse micronodular and hazy pulmonary opacities with bilateral hilar fullness (see Figure 4).