case_images: PMC11735002_01
case_images: A, B, and C showing three annular erythematous plaques over the scalp. D shows a dermoscopic view of the lesion. A: The arrow highlights an annular erythematous plaque over the temporoparietal side of the scalp. B: The arrow highlights an erythematous plaque over the temporal side of the scalp. C: The arrow highlights an erythematous plaque over the occipital side of the scalp. D: Dermoscopy shows arborizing blood vessels (indicated by the arrow) and non-scarring alopecia without epidermal changes. Intact hair follicles, free of obstruction or keratinous plugging, can be seen. Although hair follicles appear intact in the affected regions, the alopecia is due to inflammation associated with TLE, which does not lead to permanent follicular damage.
case_images: cureus-0017-00000077085-i01.jpg
case_images: PMC11735002_01_cureus-0017-00000077085-i01.jpg
case_images: FIG1
case_images: These plaques were associated with non-scarring alopecia and hair miniaturization over the frontal and vertex areas (Figure 1A, 1B, 1C). The interfollicular spaces appeared normal, without scaling, ulceration, or atrophy (Figure 1D).
case_images: A skin punch biopsy . A: H&E stain showing superficial (red arrow), deep perivascular (blue arrow), and perifollicular (black arrow) lymphocytic inflammation (4X). B: Colloidal iron stain shows increased interstitial dermal mucin (arrowheads) (20X).
case_images: cureus-0017-00000077085-i02.jpg
case_images: PMC11735002_01_cureus-0017-00000077085-i02.jpg
case_images: FIG2
case_images: A skin punch biopsy (Figure 2A, 2B) revealed superficial and deep perivascular and periadnexal lymphocytic infiltrates, dermal mucin, and edema.