case_images: PMC10757442_01
case_images: The first brain magnetic resonance image (MRI) (June 30, 2021) showed that the two lateral ventricles, third and fourth ventricles and anterior pontine cistern were slightly widened, with mild traffic hydrocephalus. Figure 1a and Figure 1b show that there were no abnormal signals in the brain parenchyma in T2-weighted images.
case_images: 10.1177_03000605231218620-fig1.jpg
case_images: PMC10757442_01_10.1177_03000605231218620-fig1.jpg
case_images: fig1-03000605231218620
case_images: The first brain magnetic resonance image (MRI) scan showed that the two lateral ventricles, third and fourth ventricles and anterior pontine cistern were slightly widened, with mild traffic hydrocephalus (Figure 1).
case_images: A second brain magnetic resonance image (MRI) with magnetic resonance venography (MRV) was taken 20 days later (July 20, 2021) and showed multiple abnormal signals in the bilateral frontal, parietal, temporal, and occipital cortices, as well as in the subcortical white matter and the right side of the pons. These features suggested reversible posterior brain injury or osmotic demyelinating syndrome. Figure 2a shows multiple abnormal signals newly detected in the temporal lobe in T2-weighted image. Figure 2b shows multiple abnormal signals newly detected in the parietal and occipital cortices in a fluid-attenuated inversion recovery (FLAIR) sequence.
case_images: 10.1177_03000605231218620-fig2.jpg
case_images: PMC10757442_01_10.1177_03000605231218620-fig2.jpg
case_images: fig2-03000605231218620
case_images: A second brain MRI scan with magnetic resonance venography (MRV) was taken and showed multiple abnormal signals in the bilateral frontal, parietal, temporal and occipital cortices, as well as in the subcortical white matter and the right side of the pons (Figure 2).
case_images: A third brain magnetic resonance image (MRI) taken seven days later (July 27, 2021) showed bilateral parietooccipital, frontal, and temporal lobes with newly added abnormal signals that suggested cerebral haemorrhage and partial hematoma formation. Figure 3a show cerebral haemorrhage and partial hematoma formation in bilateral parietooccipital, frontal, and temporal lobes in a T1-weighted image. Figure 3b show cerebral haemorrhage and partial hematoma formation in bilateral parietooccipital, frontal, and temporal lobes in a T2-weighted image.
case_images: 10.1177_03000605231218620-fig3.jpg
case_images: PMC10757442_01_10.1177_03000605231218620-fig3.jpg
case_images: fig3-03000605231218620
case_images: A third brain MRI scan showed evidence of a cerebral haemorrhage and hematoma (Figure 3).
case_images: A fourth brain MRI taken at two-year follow-up (March 27, 2022) showed abnormal signals in both parietal and occipital lobes, caused by the haematoma formation, but the scan was similar to that taken two years previously, with a reduced T1-weighted image signal. Figure 4a show that the haematoma signals of parietal and occipital lobes were reduced in a T1-weighted image. Figure 4b show that the haematoma signals of parietal and occipital lobes were reduced in a T2-weighted image.
case_images: 10.1177_03000605231218620-fig4.jpg
case_images: PMC10757442_01_10.1177_03000605231218620-fig4.jpg
case_images: fig4-03000605231218620
case_images: A fourth brain MRI taken at this time, showed abnormal signals in both parietal and occipital lobes, caused by the hematoma formation, but the scan was similar to that taken two years previously with a reduced T1-weighted image signal (Figure 4).