case_images: PMC3085384_01
case_images: Urodynamic study performed on patient (2008) showing detrusor hyperreflexia prior to voiding. PV: vesicular pressure, PR: abdominal pressure, PV-PR: detrusor pressure, Vol: volume in bladder.
case_images: CRIM2011-289389.001.jpg
case_images: PMC3085384_01_CRIM2011-289389.001.jpg
case_images: fig1
case_images: In 2008, the studies revealed OAB with DSD and OD (Figure 1).
case_images: Urodynamic study performed on patient (2010) displaying detrusor arreflexia. Pves: vesicular pressure, Pabd: abdominal pressure, Pdet: detrusor pressure, Vur: urine volume, Vinf: infusion volume.
case_images: CRIM2011-289389.002.jpg
case_images: PMC3085384_01_CRIM2011-289389.002.jpg
case_images: fig2
case_images: However, studies in 2010 revealed DA and an inability to void during pressure-flow studies (Figure 2), consistent with the patient's loss of urgency and continued incontinence.
case_images: Urinary sonogram performed on patient (2010) revealing grade II hydronephrosis consistent with noncontractile bladder.
case_images: CRIM2011-289389.003.jpg
case_images: PMC3085384_01_CRIM2011-289389.003.jpg
case_images: fig3
case_images: A urinary sonogram performed in 2010 notes a postvoid residual volume of 357 cm3 and bilateral, grade II hydronephrosis with communicative hypoechoic areas observed in both kidneys (Figure 3); this is consistent with the effects of overflow incontinence due to noncontractile bladder.