case_images: PMC10070290_01
case_images: Initial noncontrast axial computed tomography head scan demonstrating (a) cerebellar oedema with effacement of the fourth ventricle, (b and c) corresponding supratentorial ventriculomegaly.
case_images: SNI-14-94-g001.jpg
case_images: PMC10070290_01_SNI-14-94-g001.jpg
case_images: F1
case_images: An urgent computed tomography head scan demonstrated mild hydrocephalus and possible diffuse cerebellar edema, but no mass lesion or hemorrhage [Figure 1].
case_images: Initial magnetic resonance imaging showing T2/fluid attenuated inversion recovery hyperintensities in the cerebellum and brainstem (a, b, and g) and dilated lateral ventricles with periventricular hyperintensity (c and d). No infarction on diffusion-weighted imaging (e) or enhancing lesion on contrast-enhanced T1 imaging (f).
case_images: SNI-14-94-g002.jpg
case_images: PMC10070290_01_SNI-14-94-g002.jpg
case_images: F2
case_images: MRI demonstrated T2 hyperintensity involving the cerebellum, midbrain, pons, and medulla [Figure 2].
case_images: Magnetic resonance imaging 6 weeks later demonstrating resolution of signal change and swelling of the infratentorial structures and improvement of hydrocephalus (a and c). Occasional T2 hyperintensities in supratentorial white matter persist, presumably due to mild small vessel disease (b).
case_images: SNI-14-94-g003.jpg
case_images: PMC10070290_01_SNI-14-94-g003.jpg
case_images: F3
case_images: Repeat MRI imaging 6 weeks post presentation (and before discharge) demonstrated resolution of the posterior fossa edema and hydrocephalus [Figure 3].