case_images: PMC8710692_01
case_images: Brain MRI of the patient at her first episode of disease, at 8 years and 9 months. The white matter (WM) showed hyperintense in the T2-weighted axial (A-C) and FLAIR axial (D-F) views, and restricted diffusion in the diffusion-weighted imaging (G-J).
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case_images: PMC8710692_01_fped-09-801719-g0001.jpg
case_images: F1
case_images: Brain MRI showed bilateral, symmetric signal abnormalities in the white matter (WM) of the centrum semiovale and the periventricular regions, signal abnormalities in the splenium of the corpus callosum, and the head of the caudate nucleus (Figure 1). The WM showed hyperintense in the T2-weighted axial (Figures 1A-C) and FLAIR axial (Figures 1D-F) views, and restricted diffusion in the diffusion-weighted imaging (Figures 1G-J).
case_images: Brain MRI performed 45 days after the first attack. Results showed almost complete regression of the WM abnormalities (compared with Figure 1).
case_images: fped-09-801719-g0002.jpg
case_images: PMC8710692_01_fped-09-801719-g0002.jpg
case_images: F2
case_images: Brain MRI was performed 45 days later, and the WM abnormalities were almost completely subsided (Figures 2A-I).
case_images: Brain MRI of the patient at the second episode of disease 2 years later. WM appeared hyperintense in T2-weighted axial (A-C) and FLAIR axial (D-F) views, which was similar with that observed during the first attack.
case_images: fped-09-801719-g0003.jpg
case_images: PMC8710692_01_fped-09-801719-g0003.jpg
case_images: F3
case_images: The brain MRI of WM in the T2-weighted axial and FLAIR axial views were similar to that observed in the first acute episode (Figure 3).