case_images: PMC8721405_01
case_images: Imaging study. (a) Abdominal CT findings showing shrunken liver and gall bladder wall thickening with massive ascites. (b) Normal portal vein flow on Doppler ultrasonography (estimated velocity = 21.3 cm/s).
case_images: 10.1177_20406207211066176-fig1.jpg
case_images: PMC8721405_01_10.1177_20406207211066176-fig1.jpg
case_images: fig1-20406207211066176
case_images: Table 1). The CT scan showed a shrunken liver without splenomegaly and a moderate amount of ascites (Figure 1(a)), which were interpreted as liver cirrhosis following chronic hepatic GVHD by the radiologist and hepatologist rather than the expected VOD/SOS findings. In addition, the Doppler ultrasonography indicated well-preserved triphasic portal vein flow with a velocity of 21.3 cm/s (Figure 1(b)).
case_images: Post-hepatectomy histopathologic findings consistent with VOD/SOS and chronic hepatic GVHD. (a) Multifocal dilated sinusoids with hepatocyte atrophy (checked arrow) with extensive extravasation of red blood cells (solid arrow) showing hemorrhage (x100). (b) Cholestasis and bile duct dystrophy (arrow) without evidence of portal fibrosis (x400).
case_images: 10.1177_20406207211066176-fig2.jpg
case_images: PMC8721405_01_10.1177_20406207211066176-fig2.jpg
case_images: fig2-20406207211066176
case_images: We also observed some bile duct dystrophy and cholestasis which was considered findings of hepatic GVHD (Figure 2).