case_images: PMC3535733_01
case_images: Chest radiograph with remarkable reduction of lung volume as well as increased lung markings.
case_images: CRIM.PULMONOLOGY2012-678249.001.jpg
case_images: PMC3535733_01_CRIM.PULMONOLOGY2012-678249.001.jpg
case_images: fig1
case_images: Chest radiography was performed which is shown in Figure 1.
case_images: HRCT depicting diffuse areas of pulmonary infiltration, a bilateral geographic distribution of ground glass opacity and consolidation in the more dependent lung with associated traction bronchiectasis.
case_images: CRIM.PULMONOLOGY2012-678249.002.jpg
case_images: PMC3535733_01_CRIM.PULMONOLOGY2012-678249.002.jpg
case_images: fig2
case_images: Her chest CT scan (Figure 2) revealed areas of ground glass attenuation and consolidation in bilateral bases of her lungs accompanied with traction bronchiectasis.
case_images: Lung biopsy reveals scattered hyaline membranes lining alveolar septa that are thickened by interstitial edema and inflammatory cell infiltration besides hyperplasia of type II pneumocytes.
case_images: CRIM.PULMONOLOGY2012-678249.003.jpg
case_images: PMC3535733_01_CRIM.PULMONOLOGY2012-678249.003.jpg
case_images: fig3
case_images: Percutaneous lung biopsy was performed and the slides (Figure 3) showed diffuse alveolar damage (DAD) associated with hyaline membrane formation, pulmonary interstitial edema, and immature collagen edema, and focal type II pneumocyte hyperplasia were also visible.