case_images: PMC6313991_01
case_images: (a) Computed tomographic scan showed a focal lesion in the white matter of the left cerebral hemisphere. (b) FLAIR magnetic resonance imaging sequences showing lesion with surrounding cerebral edema that involved posterior central cerebral convolution, posterior limb of internal capsule, corpus callosum of the left hemisphere, and right central cerebral convolution.
case_images: CRIHEM2018-2741939.001.jpg
case_images: PMC6313991_01_CRIHEM2018-2741939.001.jpg
case_images: fig1
case_images: For this reason, a computed tomography (CT) scan was performed, and it highlighted a focal lesion with enhanced border, measuring 15 mm, in the white matter of the left cerebral hemisphere (Figure 1(a)). Magnetic resonance imaging (MRI) of the brain confirmed the presence of a lesion with surrounding cerebral edema that involved posterior central cerebral convolution, posterior limb of internal capsule, corpus callosum of the left hemisphere, and right central cerebral convolution (Figure 1(b)).
case_images: Positron emission tomography scan showing hypermetabolism in the left paraventricular region of the frontal lobe.
case_images: CRIHEM2018-2741939.002.jpg
case_images: PMC6313991_01_CRIHEM2018-2741939.002.jpg
case_images: fig2
case_images: A positron emission tomogram (PET) scan also showed fludeoxyglucose (FDG) uptake in the left lateral paraventricular region (SUVmax 11.2) (Figure 2).
case_images: Summary of APL and PCNSL treatment dates.
case_images: CRIHEM2018-2741939.003.jpg
case_images: PMC6313991_01_CRIHEM2018-2741939.003.jpg
case_images: fig3
case_images: Detailed dates of APL and PCNSL treatments are summarized in Figure 3.