case_images: PMC11700400_01
case_images: Electrocardiogram showing sinus bradycardia (48 bpm), prolongation of QT interval, ST segment depression with U waves, and T wave inversion at 2 h post-ingestion.
case_images: 10.1177_2050313X241311316-fig1.jpg
case_images: PMC11700400_01_10.1177_2050313X241311316-fig1.jpg
case_images: fig1-2050313X241311316
case_images: T wave inversion (Figure 1). The patient was diagnosed with acute barium carbonate poisoning. After his admission to ICU, the patient deteriorated further by profound hypokalemia (K+ 1.63 mmol/L) and metabolic acidosis (pH = 7.314) (Figure 2 and Table 1), managed with rapid administration of potassium chloride via the central line (23 mmol/L per hour) continuously monitoring.
case_images: Changes in serum potassium, sodium, and creatine concentrations relative to the time post-ingestion.
case_images: 10.1177_2050313X241311316-fig2.jpg
case_images: PMC11700400_01_10.1177_2050313X241311316-fig2.jpg
case_images: fig2-2050313X241311316
case_images: After his admission to ICU, the patient deteriorated further by profound hypokalemia (K+ 1.63 mmol/L) and metabolic acidosis (pH = 7.314) (Figure 2 and Table 1), managed with rapid administration of potassium chloride via the central line (23 mmol/L per hour) continuously monitoring.
case_images: Chest X-ray showing a large radiopaque mass in the stomach at 7 h (a), less dispersed radiopaque substances at 37 h post-ingestion (b), and absent at 66 h (c) post-ingestion.
case_images: 10.1177_2050313X241311316-fig3.jpg
case_images: PMC11700400_01_10.1177_2050313X241311316-fig3.jpg
case_images: fig3-2050313X241311316
case_images: Chest radiograph showed a large high-density mass in the stomach, suspicious of insoluble barium sulfate compounds (Figure 3).