case_images: PMC5903026_01
case_images: Computed tomography (CT) of the head noncontrast on day of admission: (A) right occipital hypodensity (white arrow) and bilateral occipital sulcal effacement (black arrows) and (B) bilateral cerebellar hemorrhagic infarcts (black arrows).
case_images: 10.1177_2329048X18770576-fig1.jpg
case_images: PMC5903026_01_10.1177_2329048X18770576-fig1.jpg
case_images: fig1-2329048X18770576
case_images: A noncontrast computed tomography of the head obtained on admission showed bilateral occipital sulcal effacement, right occipital subacute infarct, and bilateral cerebellar hemorrhages (Figure 1).
case_images: Magnetic resonance imaging (MRI) of the brain at supratentorial level (A-C) revealing (A) diffuse hyperintense white matter on diffusion-weighted images, (B) isointense apparent diffusion coefficient, and (C) sulcal effacement over bilateral posterior quadrant (arrow). D, T2 axial at level of pons showing hyperintensities in left central pons (arrow) and bilateral cerebellum hemispheres. At 3-month follow-up (E-F): (E) T2 axial showing right occipital encephalomalacia (arrow) and resolution of left central pons hyperintensity and (F) T2 axial showing bilateral posterior periventricular white matter hyperintensities (arrow).
case_images: 10.1177_2329048X18770576-fig2.jpg
case_images: PMC5903026_01_10.1177_2329048X18770576-fig2.jpg
case_images: fig2-2329048X18770576
case_images: Brain magnetic resonance imaging (MRI) with and without contrast was obtained on day 5 of admission, which showed (Figure 2A-D) diffuse hyperintense supratentorial white matter on diffusion-weighted images with isointense apparent diffusion coefficient, a patchy area of restricted diffusion in the right parieto-occipital region, and small hemorrhagic infarcts in the bilateral cerebellar hemispheres. A follow-up MRI brain imaging at 3 months showed T2 hyperintensities in the bilateral posterior periventricular white matter, a cortical area of encephalomalacia and gliosis in the right occipital region, and hemosiderin staining in the bilateral cerebellar hemisphere (Figure 2E and F).