case_images: PMC4862289_01
case_images: Axial (a) and sagittal (b) T1-weighted magnetic resonance imaging scans revealed a heterogeneous osteolytic mass in the right parietal region. The lesion intensely enhanced after contrast administration in the axial (c) and sagittal (d) T1-weighted magnetic resonance imaging scans
case_images: JPN-11-52-g001.jpg
case_images: PMC4862289_01_JPN-11-52-g001.jpg
case_images: F1
case_images: No other lesions were noted, and no intracranial abnormality was detected [Figure 1].
case_images: Selected axial sections (a-c) of cranial computed tomography scans demonstrating the skull defect with associated large subgaleal and extradural hematoma
case_images: JPN-11-52-g002.jpg
case_images: PMC4862289_01_JPN-11-52-g002.jpg
case_images: F2
case_images: Photomicrograph showing polymorphic infiltrate of Langerhans histiocytes (polygonal cells with kidney-shaped and elongated nuclei had longitudinal groove) admixed with many eosinophils
case_images: JPN-11-52-g003.jpg
case_images: PMC4862289_01_JPN-11-52-g003.jpg
case_images: F3
case_images: The neoplastic histiocytes showed kidney-shaped and elongated nuclei with a longitudinal groove (coffee beans like), consistent with Langerhans histiocytes [Figure 3].
case_images: Six-year follow computed tomography scan without (a) and with contrast (b) demonstrating no recurrence and spontaneous bone formation and closure of the skull defect
case_images: JPN-11-52-g004.jpg
case_images: PMC4862289_01_JPN-11-52-g004.jpg
case_images: F4
case_images: CT scan at 6-year follow-up showed no recurrence of the lesion and spontaneous bone formation and closure of previous skull defect complete resolution of the [Figure 4].