case_images: PMC8365009_01
case_images: EEG recording, showing low-voltage background activity (black arrow), theta slow-wave (red arrow), and synchronized multi-spike activity predominately in the left hemisphere (yellow arrow).
case_images: 10.1177_11795476211037782-fig1.jpg
case_images: PMC8365009_01_10.1177_11795476211037782-fig1.jpg
case_images: fig1-11795476211037782
case_images: Electroencephalography (EEG) results showed local onset paroxysmal activity in the left hemisphere with inhibited background activity (Figures 1 and 2).
case_images: EEG, showing background activity abnormality (black arrow), theta slow-wave, diffuse delta wave (blue arrow), and right hemisphere-dominant multi-spike activity (yellow arrow).
case_images: 10.1177_11795476211037782-fig2.jpg
case_images: PMC8365009_01_10.1177_11795476211037782-fig2.jpg
case_images: fig2-11795476211037782
case_images: Electroencephalography (EEG) results showed local onset paroxysmal activity in the left hemisphere with inhibited background activity (Figures 1 and 2).
case_images: Results of fluorescent staining showed positivity to AMPAR antibodies, with negative results for antibodies against NMDAR, LGI1, GABABR, and CASPR2 (Control: control sample).
case_images: 10.1177_11795476211037782-fig3.jpg
case_images: PMC8365009_01_10.1177_11795476211037782-fig3.jpg
case_images: fig3-11795476211037782
case_images: CSF analysis showed 8 white blood cells/mm3 and normal protein and glucose levels. CSF and blood were examined using indirect immunofluorescence to detect 5 antibodies known to cause autoimmune encephalitis, including those against NMDAR, leucine-rich glioma-inactivated 1 (LGI1), contactin-associated protein-like 2 (CASPR2), gamma-aminobutyric acid B receptor (GABABR), and AMPAR (Figure 3).