case_images: PMC8631422_01
case_images: Patient 1. Light microscopy shows an intact glomerulus without hypercellularity or thickened glomerular capillary walls (A). By electron microscopy, there is prominent widening of the subendothelial space by edema, cell debris, and amorphous proteinaceous deposits (B). X10,000.
case_images: fimmu-12-780107-g001.jpg
case_images: PMC8631422_01_fimmu-12-780107-g001.jpg
case_images: f1
case_images: TMA was diagnosed (see  Table 4  and  Figure 1 ) and subsequent assessment for MAHA and secondary causes of TMA was unremarkable.
case_images: PMC8631422_02
case_images: Patient 2. Light microscopy shows an intact glomerulus without hypercellularity or thickened glomerular capillary walls (A). By electron microscopy, there is prominent widening of the subendothelial space by edema, cell debris, and amorphous proteinaceous deposits. Partial basement membrane reduplication is present (B). X8,000.
case_images: fimmu-12-780107-g002.jpg
case_images: PMC8631422_02_fimmu-12-780107-g002.jpg
case_images: f2
case_images: TMA was diagnosed (see  Table 4  and  Figure 2 ) and subsequent assessment for MAHA, underlying genetic mutations, and secondary causes of TMA was unremarkable.
case_images: PMC8631422_03
case_images: Patient 3. Light microscopy shows an intact glomerulus with prominently thickened capillary walls and segmental mesangial hypercellularity (A). By electron microscopy, there is prominent widening of the subendothelial space by edema, cell debris, and amorphous proteinaceous deposits. Basement membrane reduplication is complete (B). X8,000.
case_images: fimmu-12-780107-g003.jpg
case_images: PMC8631422_03_fimmu-12-780107-g003.jpg
case_images: f3
case_images: Table 4  and  Figure 3 ).