case_images: PMC7725134_01
case_images: Head computed tomography at (A) emergency room (ER) and (B) 24 h later after the admission. The image at ER had been normal; however, those 24 h later showed low density at the bilateral thalamus, cerebellum, and brain stem.
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case_images: PMC7725134_01_AMS2-7-e611-g001.jpg
case_images: ams2611-fig-0001
case_images: A. An HCT was normal (Fig. 1A). The second HCT showed abnormal hypodensity at both thalamus, cerebellum, and brain stem (Fig. 1B).
case_images: Head magnetic resonance imaging. (A) Diffusion-weighted imaging (DWI) and (B) fluid-attenuated inversion recovery (FLAIR) followed by the second head computed tomography. Both images show bilateral symmetric lesion of the thalamus, cerebellum, and brain stem.
case_images: AMS2-7-e611-g002.jpg
case_images: PMC7725134_01_AMS2-7-e611-g002.jpg
case_images: ams2611-fig-0002
case_images: Furthermore, the subsequently performed head MRI revealed the symmetrical high-density lesions in the same area, which indicated progressed ANE with severe edema, infarction, and necrosis (Fig. 2A,B).