case_images: PMC3420699_01
case_images: Noncontrast CT scan after 24 hours of intravenous Heparin.
case_images: CRIM.NM2011-272841.001.jpg
case_images: PMC3420699_01_CRIM.NM2011-272841.001.jpg
case_images: fig1
case_images: A second CT of the brain was obtained which revealed persistent hyperdensities in the straight sinus and deep cerebral veins, with new hypodense regions involving bilateral thalami and basal ganglia likely representing edema and venous infarction (Figure 1).
case_images: Noncontrast CT scan 1 hour after intrasinus rt-PA.
case_images: CRIM.NM2011-272841.002.jpg
case_images: PMC3420699_01_CRIM.NM2011-272841.002.jpg
case_images: fig2
case_images: Five hours after the initial angiogram, repeat CT had shown improvement in the thalamic and basal ganglia edema (Figure 2).
case_images: Noncontrast CT scan after 24 hours of intrasinus rt-PA.
case_images: CRIM.NM2011-272841.003.jpg
case_images: PMC3420699_01_CRIM.NM2011-272841.003.jpg
case_images: fig3
case_images: Another follow-up CT scan was done twelve hours later, which did not show any evidence of cortical infarction or hemorrhage (Figure 3).
case_images: MRV on admission.
case_images: CRIM.NM2011-272841.004.jpg
case_images: PMC3420699_01_CRIM.NM2011-272841.004.jpg
case_images: fig4
case_images: Initial noncontrast brain CT showed hyperdense areas along the straight and sagittal sinuses, suggestive of DCST, later confirmed by magnetic resonance venography (MRV) (Figure 4).
case_images: MRV at 1 month follow-up after rt-PA.
case_images: CRIM.NM2011-272841.005.jpg
case_images: PMC3420699_01_CRIM.NM2011-272841.005.jpg
case_images: fig5
case_images: After one month follow-up MRI and MRV revealed complete patency of the venous system without residual infarct (Figure 5).