case_images: PMC7591671_01
case_images: (A) The T2-weighted magnetic resonance tomography shows a diffuse increase of signal intensity and edema in the pons during the acute phase of central pontine myelinolysis. (B) Four weeks later, the edema has resolved, but a trident-like substance defect in the pons remains (T2 weighting).
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case_images: PMC7591671_01_fendo-11-548877-g0001.jpg
case_images: F1
case_images: Magnetic resonance tomography revealed central pontine myelinolysis (Figure 1). No signs of peripheral neuropathy or central nervous system involvement other than a persistent substance defect caused by central pontine myelinolysis could be found (Figure 1).
case_images: Histology with low-grade chronic and floride tubulointerstitial nephritis. Diffuse interstitial infiltrates (arrow). Acute tubulus damage is light to moderate and potentially reversible. * tubular lumen.
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case_images: PMC7591671_01_fendo-11-548877-g0002.jpg
case_images: F2
case_images: The consecutive renal biopsy revealed chronic and acute tubulointerstitial nephritis (Figure 2).
case_images: Timeline showing the chronological order of symptoms, diagnoses, and treatment. BGA, blood gas analysis; MRI, magnetic resonance imaging.
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case_images: PMC7591671_01_fendo-11-548877-g0003.jpg
case_images: F3
case_images: Figure 3 depicts important symptoms, diagnostics, and treatment in a timeline.