case_images: PMC4142481_01
case_images: 57-year-old male was referred to our outpatient clinic because the 12-lead ECG demonstrated left ventricular (LV) hypertrophy that was later diagnosed as due to congenital coarctation of the aorta. Transthoracic echocardiography (a) Apical four-chamber view and (b) M-mode show left ventricle hypertrophy (arrows); (c and d) suprasternal views show the narrowing in the thoracic descending aorta (arrow) and the continuous wave Doppler curve without diastolic run-off (arrow).
case_images: JCIS-4-41-g002.jpg
case_images: PMC4142481_01_JCIS-4-41-g002.jpg
case_images: F1
case_images: Transthoracic echocardiography showed a non-dilated, hypertrophic left ventricle [Figure 1a and b] with end-diastolic interventricular septal thickness of 21 mm, end-diastolic LV posterior wall thickness of 12 mm, and an estimated LV mass of 449 g (LV mass index 214 g/m2). A continuous wave Doppler examination from the suprasternal notch showed a peak systolic pressure gradient in the thoracic descending aorta of 80 mmHg without diastolic run-off [Figure 1c and d], indicating a severe obstruction at the classical site of a coarctation.
case_images: 57-year-old male was referred to our outpatient clinic because the 12-lead ECG demonstrated left ventricular (LV) hypertrophy that was later diagnosed as due to congenital coarctation of the aorta. Multislice computed tomographic angiography and three-dimensional reconstruction show the aortic coarctation.
case_images: JCIS-4-41-g003.jpg
case_images: PMC4142481_01_JCIS-4-41-g003.jpg
case_images: F2
case_images: The CT scan demonstrated that both subclavian arteries originated distal to the severe coarctation, explaining the normal BP in both arms [Figure 2].