case_images: PMC11069371_01
case_images: Continued.
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case_images: However, the optic disc was slightly elevated with marked borders in the left eye and optical coherence tomography (SD-OCT) confirmed increased peripapillary retinal nerve fiber layer thickness (RNFL) with a normal result for macular ganglion cell layer (GCL) thickness (Figure 1). Ocular examination showed an increase in left optic nerve disc edema (Figure 3), confirmed by examination of RNFL in SD-OCT (Figure 1). A further two weeks later, DBCVA was 0.02, the angle of the exotropia decreased, there was no visible disc edema (Figure 3) and RNFL was also within normal limits, but GCL reduction appeared (Figure 1). Three months after the beginning of the steroid treatment, DBCVA in the affected eye was 0.3 and only periodically did exotropia occur, no optic nerve disc edema was seen (Figure 3), however, a reduction of RNFL and GCL was observed in SD-OCT (Figure 1). At the examination of the patient's left eye one year after the ON episode, DBCVA was 0.5, exotropia occurred only in moments of lack of concentration, and optic nerve neuropathy was visible at the fundus of the eye (Figure 3), which was confirmed by RNFL and GCL reduction in SD-OCT (Figure 1).
case_images: Evaluation of ganglion cell layer (GCL) and retinal nerve fiber layer (RNFL) in Optical Coherence Tomography (OCT) of a patient with myelin oligodendrocyte glycoprotein (MOG) antibody-associated optic neuritis during 1-year follow-up.
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case_images: Pattern visual evoked potentials (PVEPs, ISCEV Standard 2016 [4]) showed slightly increased latencies of P100 waves for a large (1  4') checkerboard in both eyes and a reduction of P100 amplitude for a large (1  4') and small (0  16') checkerboards in the left eye (Figure 2). In the PVEPs, the only abnormality was a prolongation of P100 latency for a small (0  16') checkerboard (Figure 2).
case_images: Evaluation of visual evoked potentials (PVEP) of a patient with myelin oligodendrocyte glycoprotein (MOG) antibody-associated optic neuritis in the left eye during 1-year follow-up.
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case_images: Ocular examination showed an increase in left optic nerve disc edema (Figure 3), confirmed by examination of RNFL in SD-OCT (Figure 1). Two weeks after treatment began, DBCVA of the left eye improved to counting fingers at 30 centimeters and the reduction of the optic nerve edema was visible (Figure 3), however, the eye was still in exotropia. A further two weeks later, DBCVA was 0.02, the angle of the exotropia decreased, there was no visible disc edema (Figure 3) and RNFL was also within normal limits, but GCL reduction appeared (Figure 1). Three months after the beginning of the steroid treatment, DBCVA in the affected eye was 0.3 and only periodically did exotropia occur, no optic nerve disc edema was seen (Figure 3), however, a reduction of RNFL and GCL was observed in SD-OCT (Figure 1). At the examination of the patient's left eye one year after the ON episode, DBCVA was 0.5, exotropia occurred only in moments of lack of concentration, and optic nerve neuropathy was visible at the fundus of the eye (Figure 3), which was confirmed by RNFL and GCL reduction in SD-OCT (Figure 1).
case_images: Evaluation of the optic nerve disc appearance of a patient with myelin oligodendrocyte glycoprotein (MOG) antibody-associated optic neuritis during 1-year follow-up.
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case_images: Kinetic perimetry performed at that time showed a narrowing of the visual field to central 10 degrees in the left eye (Figure 4). The field of view has improved to 20 central degrees (Figure 4).