case_images: PMC6530217_01
case_images: Adamantinoma-like EWS: ill-defined lesion composed of cells arranged in lobules and nests (H and E x 100).
case_images: CRIPA2019-5158182.001.jpg
case_images: PMC6530217_01_CRIPA2019-5158182.001.jpg
case_images: fig1
case_images: Immunoreactivity for p16, WT1, chromogranin, S100, EMA, vimentin, and desmin were negative in the lesional cells (Figures 1, 2, 3, and 4).
case_images: Adamantinoma-like EWS: H&E x400.
case_images: CRIPA2019-5158182.002.jpg
case_images: PMC6530217_01_CRIPA2019-5158182.002.jpg
case_images: fig2
case_images: Immunoreactivity for p16, WT1, chromogranin, S100, EMA, vimentin, and desmin were negative in the lesional cells (Figures 1, 2, 3, and 4).
case_images: Adamantinoma-like EWS: prominent squamoid morphology with peripheral palisading. ((a) H&E X 200; (b) H&E X 400).
case_images: CRIPA2019-5158182.003.jpg
case_images: PMC6530217_01_CRIPA2019-5158182.003.jpg
case_images: fig3
case_images: Immunoreactivity for p16, WT1, chromogranin, S100, EMA, vimentin, and desmin were negative in the lesional cells (Figures 1, 2, 3, and 4).
case_images: FISH image showing separation of orange and green fluorescence indicating presence of EWSR1 translocation.
case_images: CRIPA2019-5158182.005.jpg
case_images: PMC6530217_01_CRIPA2019-5158182.005.jpg
case_images: fig5
case_images: Immunoreactivity for p16, WT1, chromogranin, S100, EMA, vimentin, and desmin were negative in the lesional cells (Figures 1, 2, 3, and 4).
case_images: RT-PCR showing ESW-FLI1 fusion product.
case_images: CRIPA2019-5158182.006.jpg
case_images: PMC6530217_01_CRIPA2019-5158182.006.jpg
case_images: fig6
case_images: EWSR testing with break-apart analysis confirmed EWSR1 and FLI-1 rearrangements (Figure 5).