case_images: PMC10313390_01
case_images: (A-C) Brain MRI of the patient in case 1 indicated high signal intensity of bilateral temporal lobes and insular cortex in a T2WI sequence performed without enhancement on a contrast-enhanced scan (10 days after onset). (D-F) Cervicothoracic MRI of the patient in case 1 showed extensive thickening of the cervical and thoracic spinal cord, especially the cervical spinal cord, and an increased signal intensity in T2WI sequence, with slightly patchy enhancement of the lesions on contrast-enhanced scan (10 days after onset).
case_images: fneur-14-1121421-g001.jpg
case_images: PMC10313390_01_fneur-14-1121421-g001.jpg
case_images: fig1
case_images: B (SSB), antinuclear antibody (ANA) and anti-neutrophil cytoplasmic antibody (ANCA) were also negative. Brain magnetic resonance imaging (MRI) indicated high signal intensity of the bilateral temporal lobes and insular cortex on a T2WI sequence without enhancement on a contrast-enhanced scan (Figures 1A-C). Cervicothoracic MRI showed symmetrical long T1 and long T2 signals with slight enhancement on a contrast-enhanced scan (Figures 1D-F).
case_images: PMC10313390_02
case_images: (A-C) Brain MRI of the patient in case 2 was normal (2 months after onset). (D-F) Spinal MRI of the patient in case 2 indicated longitudinal linear abnormity with long T2 signal in the cervical and thoracic spinal cord and was not enhanced on the contrast-enhanced scan (2 months after onset).
case_images: fneur-14-1121421-g002.jpg
case_images: PMC10313390_02_fneur-14-1121421-g002.jpg
case_images: fig2
case_images: Electromyogram and brain MRI were normal (Figures 2A-C), whereas spinal MRI indicated longitudinal linear abnormity with a long T2 signal in the cervical and thoracic spinal cord and was not enhanced on the contrast-enhanced scan (Figures 2D-F).