case_images: PMC5840949_01
case_images: Disease progression of patient from ED (t = 0 hr) presentation to cardiogenic shock (t = +9 hr).
case_images: CRIPE2018-7858192.001.jpg
case_images: PMC5840949_01_CRIPE2018-7858192.001.jpg
case_images: fig1
case_images: CMP was significant for metabolic acidosis and hyperkalemia (Figure 1). Chest X-ray was unremarkable (Figure 2).
case_images: CXR (AP) shows a prominent thymus, normal cardiothoracic ratio, sharp costophrenic angles without cardiomegaly, consolidation, or increased perivascular markings.
case_images: CRIPE2018-7858192.002.jpg
case_images: PMC5840949_01_CRIPE2018-7858192.002.jpg
case_images: fig2
case_images: CMP was significant for metabolic acidosis and hyperkalemia (Figure 1). Chest X-ray was unremarkable (Figure 2). Lower limb blood pressures at that time were undetectable (see Figure 2 for detailed exam).
case_images: Color Doppler showing narrowing (arrow) with marked flow acceleration in descending aorta. AAo: ascending aorta; DAo: descending aorta.
case_images: CRIPE2018-7858192.003.jpg
case_images: PMC5840949_01_CRIPE2018-7858192.003.jpg
case_images: fig3
case_images: Critical coarctation of the aorta (CoA) was suspected, and 2D echocardiogram confirmed severe discrete coarctation of the aorta (Figure 3).