case_images: PMC6345076_01
case_images: Analytical data: (A) Hyporegenerative microcytic anaemia, abnormality in iron metabolism (Fe), elevation of acute phase reactants and liver enzyme abnormality. (B) Morphology of peripheral blood with hypochromia, intense rouleaux and thrombocytosis. (C) Proteinogram with hypoalbuminemia and hypergammaglobulinemia.
case_images: can-12-882fig1.jpg
case_images: PMC6345076_01_can-12-882fig1.jpg
case_images: figure1
case_images: The analytical and peripheral blood morphology data are shown in Figure 1.
case_images: Abdominal magnetic nuclear resonance: (A) Axial (A1) and coronal (A2) view during diagnosis: increased liver size, with finely heterogeneous parenchyma and no focal lesions. (B) Axial (B1) and coronal (B2) view after treatment: liver size within normal limits with homogeneous parenchyma.
case_images: can-12-882fig2.jpg
case_images: PMC6345076_01_can-12-882fig2.jpg
case_images: figure2
case_images: During magnetic nuclear resonance (MNR), we observed severe hepatomegaly suggestive of infiltration, with increased retroperitoneal adenopathies and compromise of the right renal excretory duct (Figure 2). Radiological data (CT and MRI scans) confirmed this regression of peliosis (Figure 2).
case_images: Peliosis hepatis. Cystic spaces with no endothelial lining (arrow). HE x 10.
case_images: can-12-882fig3.jpg
case_images: PMC6345076_01_can-12-882fig3.jpg
case_images: figure3
case_images: The liver biopsy shows slight portal fibrosis, marked dilation of the sinusoids that appear filled with blood material and with a tendency to form cystic cavities, compatible with peliosis hepatis (Figure 3).