case_images: PMC5546119_01
case_images: ECG.
case_images: CRIC2017-8458054.001.jpg
case_images: PMC5546119_01_CRIC2017-8458054.001.jpg
case_images: fig1
case_images: ST elevation in anterolateral leads (Figure 1) and troponins I peak to 38.97 ng/mL.
case_images: Echocardiogram showing apical hypokinesia.
case_images: CRIC2017-8458054.002.jpg
case_images: PMC5546119_01_CRIC2017-8458054.002.jpg
case_images: fig2
case_images: Echocardiogram (Figure 2) showed apical hypokinesia, ejection fraction of 40%, and systolic anterior motion of mitral valve with hyperdynamic left ventricle, in the absence of intracoronary thrombus formation in the angiogram (Figure 3). Subsequently patient underwent coronary angiography which showed no evidence of CAD, hypokinetic apex, and ejection fraction of 55%, which were consistent with TC. Electroencephalogram (EEG) showed evidence of generalized tonic-clonic seizure.
case_images: Coronary angiogram and left ventriculography.
case_images: CRIC2017-8458054.003.jpg
case_images: PMC5546119_01_CRIC2017-8458054.003.jpg
case_images: fig3
case_images: Echocardiogram (Figure 2) showed apical hypokinesia, ejection fraction of 40%, and systolic anterior motion of mitral valve with hyperdynamic left ventricle, in the absence of intracoronary thrombus formation in the angiogram (Figure 3). Subsequently patient underwent coronary angiography which showed no evidence of CAD, hypokinetic apex, and ejection fraction of 55%, which were consistent with TC. Electroencephalogram (EEG) showed evidence of generalized tonic-clonic seizure.