case_images: PMC5367259_01
case_images: (a) Kaolin-activated thromboelastography (TEG) and (b) tissue factor-activated TEG tracings from an azotemic cat with bilateral perinephric pseudocysts and factor XII (FXII) deficiency; (c) kaolin-activated TEG and (d) tissue factor-activated TEG tracings from a normal cat with FXII deficiency. Part (d) is considered a normal tissue factor-activated tracing
case_images: 10.1177_2055116915585025-fig1.jpg
case_images: PMC5367259_02_10.1177_2055116915585025-fig1.jpg
case_images: fig1-2055116915585025
case_images: Figure 1c), whereas the TF-activated TEG was normal (Table 1; Figure 1d). FXII activity was 7%, consistent with a FXII deficiency.
case_images: Kaolin-activated thromboelastography from a normal cat with normal partial thromboplastin time and presumed normal factor XII concentration
case_images: 10.1177_2055116915585025-fig2.jpg
case_images: PMC5367259_01_10.1177_2055116915585025-fig2.jpg
case_images: fig2-2055116915585025
case_images: Kaolin-activated TEG exhibited delayed clot formation characterized by markedly prolonged reaction (R) and kappa (kappa) times, and a decreased angle compared with normal (Table 1; Figures 1a and 2), whereas the TF-activated TEG revealed a normal to mildly hypercoagulable tracing characterized by a mildly increased maximum amplitude (Table 1; Figure 1b).
case_images: PMC5367259_02
case_images: (a) Kaolin-activated thromboelastography (TEG) and (b) tissue factor-activated TEG tracings from an azotemic cat with bilateral perinephric pseudocysts and factor XII (FXII) deficiency; (c) kaolin-activated TEG and (d) tissue factor-activated TEG tracings from a normal cat with FXII deficiency. Part (d) is considered a normal tissue factor-activated tracing
case_images: 10.1177_2055116915585025-fig1.jpg
case_images: PMC5367259_02_10.1177_2055116915585025-fig1.jpg
case_images: fig1-2055116915585025
case_images: Figure 1c), whereas the TF-activated TEG was normal (Table 1; Figure 1d). FXII activity was 7%, consistent with a FXII deficiency.