case_images: PMC11829613_01
case_images: Papular lesions in the healing phase on the patient's back
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case_images: FIG1
case_images: Other notable findings during the physical exam included white lesions on the tongue, palate, and oropharynx, compatible with oral candidiasis, and papular lesions in the healing phase in his back (Figure 1).
case_images: High-resolution CT scan of the lungs showing the bilateral ground-glass opacities (red arrows). CT: computed tomography
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case_images: FIG2
case_images: A high-resolution chest CT scan revealed bilateral ground-glass opacities (Figure 2).
case_images: Bronchofibroscopy showing no lesions
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case_images: FIG3
case_images: BAL (Figure 3) fluid analysis was positive for Pneumocystis jirovecii (by PCR), Cryptococcus neoformans (by culture), and multidrug-resistant Escherichia coli (by culture).
case_images: Coronal (A) and axial (B) view of the cerebral CT scan showing the hypodense area on the left gangliocapsular region (red arrows). CT: computed tomography
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case_images: FIG4
case_images: CT scan was performed, showing hypodense areas in the left gangliocapsular region with mass effect (Figure 4A-4B). Cerebral MRI confirmed the presence of tumefactive lesions in the head of the right caudate nucleus and in the head and body of the left caudate nucleus, with hypersignal in
case_images: Axial view of the cerebral MRI T2 FSE showing the tumefactive lesions on the caudate nucleus bilaterally (red arrows). MRI: magnetic resonance imaging, FSE: fast spin-echo
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case_images: FIG5
case_images: T2. These lesions were compressing the frontal horns of the lateral ventricles (Figure 5A-5B) and were suggestive of cryptococcomas.
case_images: Axial view of the cerebral CT scan showing (A) hyperdensity in the corpus striatum, internal capsule, thalamus, and intraventricular area compatible with hemorrhage and (B) hemorrhagic lesion with signs of hydrocephalus. CT: computed tomography
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case_images: FIG6
case_images: Emergent cerebral CT showed left gangliocapsular hyperdensity, compatible with hemorrhagic transformation following ischemic stroke, with ipsilateral intraventricular bleeding and hydrocephalus (Figure 6A-6B).