case_images: PMC5902092_01
case_images: (a) Transverse view of abdominal CT shows periaortic soft tissue thickening adjacent to aorta (arrow). (b) Coronal view of abdominal CT without IV contrast shows disruption of calcification in aortic wall (upper arrow) and periaortic soft tissue thickening around the infrarenal aorta and around bifurcation of aorta into common iliac arteries (lower arrow). (c) Sagittal view of abdominal CT, with IV contrast, shows pseudoaneurysm formation (upper arrow) and periaortic soft tissue thickening around the infrarenal aorta (lower arrow). (d) Reconstructive CT angiogram shows pseudoaneurysm formation (arrow).
case_images: CRIM2018-6845617.001.jpg
case_images: PMC5902092_01_CRIM2018-6845617.001.jpg
case_images: fig1
case_images: Follow-up abdominal CT one week later showed progressive inflammatory changes around infrarenal aorta extending to common iliac arteries and proximal external and internal iliac arteries, mild mural thickening of proximal abdominal aorta, and development of penetrating intimal ulcers, suggesting progression of aortitis (Figures 1(a)-1(d)).
case_images: Low-power photomicrograph of resected aortic segment with hematoxylin and eosin stain shows inflammation of aortic wall. Inset: high-power photomicrograph of resected aortic segment with hematoxylin and eosin stain shows abundant neutrophylic infiltration of aortic wall without detection of the microorganism.
case_images: CRIM2018-6845617.002.jpg
case_images: PMC5902092_01_CRIM2018-6845617.002.jpg
case_images: fig2
case_images: Histopathology of the resected aorta revealed severe neutrophylic infiltration and other inflammatory cells within the arterial wall, without evident bacteria (Figure 2).