case_images: PMC6119709_01
case_images: Bilateral, hypodense basal ganglia necrosis in unenhanced CT (arrows); Philips Ingenuity 5 mm.
case_images: fneur-09-00708-g0001.jpg
case_images: PMC6119709_01_fneur-09-00708-g0001.jpg
case_images: F1
case_images: An unenhanced computed tomography (CT) of the head showed almost symmetrical bilateral hypointensities of the globus pallidus (Figure 1).
case_images: Bilateral basal ganglia necrosis with T2w hyperintense alterations (A) and hemoside deposits (B). These changes are diffusion-disturbed (C,D). 3 T Philips Ingenia, (A) FLAIR, (B) SWI, (C) b1000 image, (D) ADC map.
case_images: fneur-09-00708-g0002.jpg
case_images: PMC6119709_01_fneur-09-00708-g0002.jpg
case_images: F2
case_images: MRI of the brain 3 weeks after hospitalization confirmed the bilateral lesions of the globus pallidus seen on CT, characterized by restricted diffusion and FLAIR-hyperintense signal changes (Figure 2).
case_images: Planar T2w hyperintense signal changes of the entire white matter (A) with diffusion restriction (B,C) 3 T Philips Ingenia, (A) FLAIR, (B) b1000 Figure, (C) ADC Map.
case_images: fneur-09-00708-g0003.jpg
case_images: PMC6119709_01_fneur-09-00708-g0003.jpg
case_images: F3
case_images: A follow-up MRI-scan of the brain 5 weeks after the initial event showed, in addition to the known changes of the basal ganglia, a symmetrical, extensive increased FLAIR-signal with correlating marked diffusion restrictions of the white matter, primarily in the fronto-parietal regions of both hemispheres (Figure 3).
case_images: Slightly T2w hyperintense signal changes of the white matter (A) without diffusion restriction (B,C), Atophy of the parenchyma with slightly wider inner CSF. 3 T Philips Ingenia, (A) FLAIR, (B) b1000 Figure, (C) ADC Map. Overall there is a marked regression of the imaging findings.
case_images: fneur-09-00708-g0004.jpg
case_images: PMC6119709_01_fneur-09-00708-g0004.jpg
case_images: F4
case_images: A slight symmetrical enlargement of the ventricles could be seen indicating a mild atrophy (Figure 4).