case_images: PMC11787821_01
case_images: Prior EKG. This is an EKG taken prior to the first appearance of electrical changes due to acute pericarditis.
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case_images: FIG1
case_images: In comparison to a prior EKG (Figure 1), follow-up EKG the next morning (Figure 2) showed widespread ST segment elevations and PR interval depressions suggesting pericarditis.
case_images: EKG showing acute pericarditis. EKG showing normal sinus rhythm with widespread ST segment elevations (blue arrows) and PR interval depressions (yellow arrows), which was more apparent when comparing it to previous normal EKGs. Both changes were easier to identify when compared to previous EKGs.
case_images: cureus-0017-00000076861-i02.jpg
case_images: PMC11787821_01_cureus-0017-00000076861-i02.jpg
case_images: FIG2
case_images: In comparison to a prior EKG (Figure 1), follow-up EKG the next morning (Figure 2) showed widespread ST segment elevations and PR interval depressions suggesting pericarditis.
case_images: Echocardiogram showing pericardial effusion. Echocardiogram showing pericardial effusion (yellow arrow). Fluid collection was followed throughout the hospital stay to determine resolution of the pericardial effusion.
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case_images: FIG3
case_images: An echocardiogram was performed on day 2, demonstrating severely increased concentric wall thickness and large pericardial effusion (Figure 3) without hemodynamic compromise.
case_images: Post-procedure echocardiogram. Echocardiogram taken a week following the pericardiocentesis, prior to discharge.
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case_images: FIG4
case_images: Echocardiograms performed on hospital days 8 and 9 (Figure 4), which showed mild pericardial effusion and left pleural effusion.
case_images: Post-hospitalization cardiac MRI. Cardiac MRI taken following discharge from the hospital. The yellow arrows highlight the thickening of the pericardium.
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case_images: FIG5
case_images: Cardiac magnetic resonance imaging was performed after discharge, which showed 6mm pericardial thickness, trivial pericardial effusion, and pericardial inflammation without myocardial involvement (Figure 5).