case_images: PMC9489477_01
case_images: Autopsy findings showing herniated stomach in the chest cavity with severe mediastinal deviation
case_images: 41935_2022_299_Fig1_HTML.jpg
case_images: PMC9489477_01_41935_2022_299_Fig1_HTML.jpg
case_images: Fig1
case_images: Upon opening the chest cavity, the stomach, markedly distended, was occupying much of the left pleural cavity (Fig. 1).
case_images: a Autopsy findings showing the diaphragm defect (arrow) and ecchymotic stomach portion (lesser curvature) entrapped in the hernia defect (b) (arrow)
case_images: 41935_2022_299_Fig2_HTML.jpg
case_images: PMC9489477_01_41935_2022_299_Fig2_HTML.jpg
case_images: Fig2
case_images: This mediastinal deviation was due to an ascension of the stomach into the chest cavity through a 2 x 1.5 cm defect in the posterior left hemidiaphragm (Fig. 2a). Further examination remarked an ecchymotic appearance of the stomach portion entrapped in the hernia defect (Fig. 2b) suggesting recent strangulation of the stomach.
case_images: Histology findings of different organs, hematoxylin and eosin (HE) coloration. a Alveolar collapse with polymorphic inflammatory infiltrate and vascular congestion (HE x 100). b Normal diaphragm muscle cells component (HE x 100). c Chronic inflammatory changes of the gastric mucosa with vascular congestion (HE x 100)
case_images: 41935_2022_299_Fig3_HTML.jpg
case_images: PMC9489477_01_41935_2022_299_Fig3_HTML.jpg
case_images: Fig3
case_images: The histology study showed alveolar collapse with the presence of a polymorphic inflammatory infiltrate of the left lung (Fig. 3a) consistent with atelectasis associated with signs of pulmonary infection. The diaphragm histology study (Fig. 3b) showed normal muscle cells component, while chronic inflammatory changes of the gastric mucosa with vascular congestion were found (Fig. 3c).