case_images: PMC10642239_01
case_images: (A,B) Brain diffusion weighted imaging and apparent diffusion coefficient, revealing acute ischemic stroke (pointed by yellow arrow) and subacute ischemic stroke (pointed by red arrow) involving the right centrum semiovale. (C) Brain magnetic resonance angiography, suggesting multiple cerebral arteriosclerosis and stenosis of cerebral artery.
case_images: fneur-14-1249464-g001.jpg
case_images: PMC10642239_01_fneur-14-1249464-g001.jpg
case_images: fig1
case_images: The head diffusion weighted imaging (DWI, Figure 1) scan showed acute infarction in the right centrum semiovale. The older infarct showed in the DWI, presented high signal intensity on ADC (pointed by red arrow in Figure 1), indicating that the lesion may be the lesion of subacute cerebral infarction and had nothing to do with the present attack.
case_images: (A-F) Fluid-attenuated inversion recovery. (G,H) Brain T1-weighted image. (H-N) T2-weighted image, revealing multiple old infarction and encephalomalacia foci combined with gliosis of bilateral frontal, parietal, occipital and temporal lobe. Virchow-Robin Spaces and demyelination was observed in the basal ganglia of both sides, and no obvious abnormalities were observed in the brainstem.
case_images: fneur-14-1249464-g002.jpg
case_images: PMC10642239_01_fneur-14-1249464-g002.jpg
case_images: fig2
case_images: Head magnetic resonance imaging (MRI, Figure 2) results revealed multiple cerebral softening lesions with gliosis in the bilateral frontal, parietal, occipital, and temporal lobes.
case_images: (A-G) No legions showed in striatum from Brain T1-weighted image sagittal images.
case_images: fneur-14-1249464-g003.jpg
case_images: PMC10642239_01_fneur-14-1249464-g003.jpg
case_images: fig3
case_images: T1W1 signal in the right striatum observed on the head MRI (Figure 3). Therefore, the possibility of hemichorea caused by NKH is ruled out.