case_images: PMC10375235_01
case_images: (A) Axial T2 FLAIR scan and diffusion-weighted imaging (DWI) revealed a high-intensity signal [apparent diffusion coefficient (ADC) shows a low signal intensity, indicating acute cerebral ischemia] in the left frontal lobe and midbody of the CC 2 days after onset. Magnetic resonance angiography (MRA) images revealed a vessel occlusion of the left A2. (B) Axial T2 FLAIR and DWI show a change to a low-intensity signal (conversely, ADC shows a high-intensity signal, indicating a chronic phase) in the left frontal lobe and the midbody of the CC 5 months after onset.
case_images: fneur-14-1203450-g0001.jpg
case_images: PMC10375235_01_fneur-14-1203450-g0001.jpg
case_images: F1
case_images: CC) on admission (Figure 1A), and a low-intensity signal in the same regions 5 months following onset (Figure 1B). Computed tomography and MRI did not reveal any hemorrhages or exudates. Magnetic resonance angiography (MRA) images revealed a vessel occlusion of the left A2 (Figure 1A).
case_images: Sagittal T2 FLAIR scan shows postinfarct necrosis in the left frontal lobe, genu, and midbody of the CC 5 months after onset (left panel). Axial diffusion tensor tractography of the CC 5 months after onset (right panel).
case_images: fneur-14-1203450-g0002.jpg
case_images: PMC10375235_01_fneur-14-1203450-g0002.jpg
case_images: F2
case_images: MRI T2-FLAIR revealed low-intensity signals and enlarged left cerebral ventricles 5 months after onset (Figure 2, left panel). DTI showed disruptions in the genu and midbody of the left CC (Figure 2, right panel), which were bilaterally connected to the frontal and parietal cortices, respectively.