case_images: PMC6778882_01
case_images: Evolution of gasometric parameters during treatment of patient.
case_images: CRIN2019-7498373.001.jpg
case_images: PMC6778882_01_CRIN2019-7498373.001.jpg
case_images: fig1
case_images: A venous-blood gas test performed 1 h later showed that pH increased to 7.10, pCO2 47 mmHg, pO2 59 mmHg, HCO3 14 mmol/L, and increased lactate levels (9.4 mmol/L) (Figure 1). Three hours after increasing intravenous fluids, a new venous-blood gas test showed persistent acidemia with a pH of 7.16, pCO2 33 mmHg, pO2 60 mmHg, HCO 11.8 mmol/L, and increased lactate levels (13.2 mmol/L), despite maintaining hemodynamic stability, with MAP >65 mmHg without vasopressor treatment, urinary output 200-300 ml/h, and being afebrile without any clinical data demonstrating tissue hypoperfusion (Figure 1). PRIS. Propofol infusion was stopped, and correction of metabolic acidosis and a progressive decrease in serum lactate levels were observed, with normalization 4 h later (pH 7.4, pCO2 32 mmHg, HCO3 20 mmol/L, and lactate 1.7 mmol/L) (Figure 1).
case_images: Image shows the patient's lipemic serum.
case_images: CRIN2019-7498373.002.jpg
case_images: PMC6778882_01_CRIN2019-7498373.002.jpg
case_images: fig2