case_images: PMC5379783_01
case_images: The patient developed ptosis, opthalmoplegia, chemosis, pupillary dilatation, and loss of sensation over the left eyelid in the immediate postoperative period
case_images: AJNS-12-116-g001.jpg
case_images: PMC5379783_01_AJNS-12-116-g001.jpg
case_images: F1
case_images: Postoperatively, the patient developed left sided ptosis, opthalmoplegia, pupillary dilatation, and loss of sensation over the left eyelid [Figure 1].
case_images: Three months after the surgery, there was partial recovery of ptosis and opthalmoplegia
case_images: AJNS-12-116-g002.jpg
case_images: PMC5379783_01_AJNS-12-116-g002.jpg
case_images: F2
case_images: After 3 months, the patient showed improvement in the deficit (partial recovery of 3rd nerve function), which ruled out vascular injury and pointed towards neuropraxic injury [Figures 2-4].
case_images: Abduction of the left eye has not recovered in three months
case_images: AJNS-12-116-g003.jpg
case_images: PMC5379783_01_AJNS-12-116-g003.jpg
case_images: F3
case_images: After 3 months, the patient showed improvement in the deficit (partial recovery of 3rd nerve function), which ruled out vascular injury and pointed towards neuropraxic injury [Figures 2-4].
case_images: Three months after the surgery, chemosis is still present while opthalmoplegia has partially recovered
case_images: AJNS-12-116-g004.jpg
case_images: PMC5379783_01_AJNS-12-116-g004.jpg
case_images: F4
case_images: After 3 months, the patient showed improvement in the deficit (partial recovery of 3rd nerve function), which ruled out vascular injury and pointed towards neuropraxic injury [Figures 2-4].