A 64-year-old man was hospitalized for end-stage renal disease. He chose PD treatment. Eighteen months ago, the patient was diagnosed with IgA nephropathy by renal biopsy and was diagnosed as having stage 5 chronic kidney disease 1 wk ago. The patient had a history of hypertension, hyperuricemia, and gout, a history of sleep apnea syndrome and pharyngopalatoplasty for treatment of sleep apnea syndrome, as well as history of laparoscopic cholecystectomy. Born in Beijing, the patient has lived here for a long time, has not been to epidemic areas and pastoral areas, and has no bad habits such as smoking and drinking. His parents suffered from hypertension and denied a history of other familial genetic diseases. The patient’s temperature was 36.2 °C, heart rate 70 bpm, respiratory rate 20 breaths/min, blood pressure 180/90 mmHg, and oxygen saturation in room air 99%. His body height was 167 cm, body weight 91 kg, and body mass index 32.6 kg/m2. Physical examination showed that the breath sound of both lungs was thick, and both lungs had scattered wet rales. He had abdominal swelling and slight bilateral symmetrical finger depression edema of lower limbs, with no tenderness or rebound pain and no percussion pain in the renal area. No other positive signs were found in the physical examination. Routine blood analysis showed that his hemoglobin was 77 g/L, platelet count 69 × 109/L, serum creatinine 1065 mmol/L, and glomerular filtration rate 4 mL/min. Urinalysis showed urinary occult blood (+) and urine protein (++). The level of B-type natriuretic peptide was 1182.90 pg/mL. Liver function and coagulation tests were normal. Abdominal computed tomography (CT) showed no clear display of gallbladder, and both kidneys had slight atrophy. Cardiac ultrasound revealed whole heart enlargement.