A 34-year-old Japanese woman suffered from headache and insomnia starting 2 days after her first childbirth. During her pregnancy, she did not experience any complications, including pregnancy-induced hypertension. However, postpartum hypertension was noted (160–170 mmHg/90–100 mmHg). She visited our clinic complaining of a visual field defect that had appeared 8 days after her headache onset. Corrected visual acuities for the right and left eye were 20/20 and 20/25, respectively. Intraocular pressure in both eyes was 15 mmHg. The anterior segments and lenses appeared normal. Multiple yellowish dots in the deep retina were noted around the optic nerve head in both eyes (indicated by the arrows in Fig. ), and resembled Elschnig’s spots and ischemic choroidal infarcts []. Optical coherence tomography (OCT) demonstrated exudative retinal detachments around the optic nerve head in both eyes. Visual field testing conducted by standard automated perimetry showed left homonymous hemianopsia. We referred the patient to a neurologist for neurological examinations regarding the headache and left homonymous hemianopsia. Results from serum biochemistry tests and a cerebrospinal fluid examination were normal. Magnetic resonance imaging (MRI) revealed edema in the basal ganglions and right occipital lobe. At day 2 after starting treatments with an infusion of a free-radical scavenger (edaravone, 60 mg per day) and anticoagulant (heparin sodium, 10,000 U per day), the left homonymous hemianopsia disappeared and MRI showed subsidence of the brain edema. The treatment was continued for 3 days, and the patient's symptoms improved. After improvement of the visual symptoms, cerebral angiography appeared normal, and the yellowish dots in the deep retina and exudative detachments around the optic nerve heads were no longer observed. There has been no recurrence of symptoms during the 4 years of follow-up. The patient gave consent for her clinical details and clinical images to be published.