A previously healthy 7-year-old girl started with progressive neck pain and headache, and after being evaluated for 2 weeks with several pediatrician consultations, progressed with a visual loss following these initial symptoms, and papilledema was observed in the ophthalmologist consultation. Subsequently, the patient was submitted for a MRI exam, which showed a partially empty sella, tortuosity of the optic nerves, and also the rectification of the retina. All of the MRI signs are common imaging findings in idiopathic intracranial hypertension (IIH)[] as well as the papilledema that is the most common and important sign in IIH, and is a result of axoplasmic flow stasis secondary to increased ICP, producing edema of the retinal nerve fibers emanating from the optic disc.[] The vision loss is correlated with the severity of papilledema.[] A LP with opening and closing pressure was performed, with continuous non-invasive ICP monitoring. Before the beginning of the procedure, the non-invasive measure showed a P2/P1 ratio of 1.1, reflecting altered brain compliance which expresses the capability to buffer an intracranial volume increase while avoiding a rise in ICP. During the procedure, the child started crying, with some loss on the quality of the signal acquired; however, the software could identify 1 min of good quality signal that showed a P2/P1 ratio of 1.38. The opening pressure was 32 cm H2O, and the closing pressure was 15 cm H2O and as a result, the child had prompt relief in her headache. Authors recommended that for children an opening pressure above 28 cm H2O should be considered as elevated ICP. Another study also considered CSF measures n28 cm H2O as “normal” for most children.[] After the procedure, the non-invasive method showed an evident normalization of ICP pulse curve morphology, with the P2/P1 ratio coming to 0.65 [].