Male patient diagnosed with rapidly progressive glomerulonephritis with anti-glomerular basement membrane antibodies, treated with steroids, cyclophosphamide and plasmapheresis without results. During the 5 years on PD he suffered 10 peritonitis, 8 due to Staphylococcus aureus. The last one was due to Candida albicans; therefore the catheter was removed and he was transferred to HD.
He was a medium-high transporter initially, becoming a high transporter. Six months later he presented with abdominal pain and a mass effect in the hypogastrium. CT scan showed peritoneal thickening, pelvic fluid collection with septa encompassing loops, and a large gastric and intestinal dilatation. The patient refused surgical treatment, required several admissions for intestinal occlusion, suffered progressive deterioration of the general condition and died shortly afterwards.

